Pathogenic Escherichia coli
MetadataShow full item record
© 2014 Elsevier Ltd. All rights reserved. Some strains of Escherichia coli cause disease of the gastrointestinal tract. A subgroup of these, the Shiga toxin-producing E. coli, which includes E. coli O157:H7, are common commensals of ruminant food animals. Numerous outbreaks of severe and often fatal human disease have been caused by these strains. Substantial work has been done to understand their clinical characteristics and ecology as well as to control them on farm and during meat processing.
Showing items related by title, author, creator and subject.
Increasing incidence of Escherichia coli bacteraemia is driven by an increase in antibiotic-resistant isolates: Electronic database study in Oxfordshire 1999-2011Schlackow, I.; Stoesser, N.; Walker, A.; Crook, D.; Peto, T.; Wyllie, D.; Bejon, P.; Berendt, T.; Bunch, C.; Finney, J.; Gearing, J.; Jones, H.; O'Connor, L.; Robinson, James; Shine, B.; Waller, D. (2012)Objectives: To investigate trends in Escherichia coli resistance, bacteraemia rates and post-bacteraemia outcomes over time. Methods: Trends in E. coli bacteraemia incidence were monitored from January 1999 to June 2011 ...
Escherichia coli strains expressing H12 antigens demonstrate an increased ability to attach to abiotic surfaces as compared with E. coli strains expressing H7 antigensGoulter, R.; Taran, E.; Gentle, I.; Gobius, K.; Dykes, Gary (2014)The role of Escherichia coli H antigens in hydrophobicity and attachment to glass, Teflon and stainless steel (SS) surfaces was investigated through construction of fliC knockout mutants in E. coli O157:H7, O1:H7 and ...
Expression and putative roles in attachment of outer membrane proteins of Escherichia coli O157 from planktonic and sessile cultureRivas, L.; Fegan, N.; Dykes, Gary (2008)Many strains of Shiga toxigenic Escherichia coli (STEC), particularly the serotype O157:H7, are foodborne pathogens causing disease in many countries throughout the world. E. coli O157:H7 is able to attach and survive on ...