Multifactorial non-cirrhotic hyperammonaemic encephalopathy
dc.contributor.author | Triplett, K. | |
dc.contributor.author | Murray, R. | |
dc.contributor.author | Anstey, Matthew | |
dc.date.accessioned | 2018-05-18T08:00:03Z | |
dc.date.available | 2018-05-18T08:00:03Z | |
dc.date.created | 2018-05-18T00:23:15Z | |
dc.date.issued | 2018 | |
dc.identifier.citation | Triplett, K. and Murray, R. and Anstey, M. 2018. Multifactorial non-cirrhotic hyperammonaemic encephalopathy. BMJ Case Reports. 2018: Article ID | |
dc.identifier.uri | http://hdl.handle.net/20.500.11937/67879 | |
dc.identifier.doi | 10.1136/bcr-2017-223245 | |
dc.description.abstract |
A 51-year-old female presented with acute confusion associated with a non-specific headache and lethargy. The patient's history included bipolar disorder on valproate and recent travel to northern Vietnam. The patient was subsequently found to have hyperammonaemia as well as a urinary tract infection and bacteraemia with Klebsiellapneumoniae. The patient was presumed to have a multifactorial non-cirrhotic hyperammonaemic encephalopathy due to a combination of a urinary tract infection and bacteraemia with K. pneumoniae, a urease-producing bacteria, and also valproate use, a medication known to interfere with ammonia elimination. The patient's treatment included supportive care, ceasing valproate, empiric then rationalised antibiotics, N-acetylcysteine and L-carnitine. We present a case of non-cirrhotic hyperammonaemic encephalopathy and explain why it is multifactorial in origin. | |
dc.publisher | British Medical Journal Publishing Group | |
dc.title | Multifactorial non-cirrhotic hyperammonaemic encephalopathy | |
dc.type | Journal Article | |
dcterms.source.volume | 2018 | |
dcterms.source.issn | 1757-790X | |
dcterms.source.title | BMJ Case Reports | |
curtin.department | School of Public Health | |
curtin.accessStatus | Fulltext not available |
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