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dc.contributor.authorLim, L.
dc.contributor.authorTran, B.
dc.contributor.authorVincan, Elizabeth
dc.contributor.authorLocarnini, S.
dc.contributor.authorWarner, N.
dc.date.accessioned2017-01-30T12:20:00Z
dc.date.available2017-01-30T12:20:00Z
dc.date.created2015-12-10T04:26:11Z
dc.date.issued2012
dc.identifier.citationLim, L. and Tran, B. and Vincan, E. and Locarnini, S. and Warner, N. 2012. HBV-related hepatocellular carcinoma: The role of integration, viral proteins and miRNA. Future Virology. 7 (12): pp. 1237-1249.
dc.identifier.urihttp://hdl.handle.net/20.500.11937/20581
dc.identifier.doi10.2217/fvl.12.113
dc.description.abstract

The development of hepatocellular carcinoma during chronic hepatitis B infection is a multifactorial process thought to be a consequence of several direct and indirect mechanisms. In this review we discuss how viral proteins and cycles of ongoing liver damage and regeneration, coupled with HBV DNA integration and aberrant miRNA expression may enhance the risk for the development of hepatocellular carcinoma. © 2012 Future Medicine Ltd.

dc.titleHBV-related hepatocellular carcinoma: The role of integration, viral proteins and miRNA
dc.typeJournal Article
dcterms.source.volume7
dcterms.source.number12
dcterms.source.startPage1237
dcterms.source.endPage1249
dcterms.source.issn1746-0794
dcterms.source.titleFuture Virology
curtin.departmentSchool of Biomedical Sciences
curtin.accessStatusFulltext not available


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