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dc.contributor.authorAshworth, Michael
dc.contributor.authorHan, H.
dc.contributor.authorKnell, G.
dc.contributor.authorPowles, S.
dc.date.accessioned2017-01-30T14:57:48Z
dc.date.available2017-01-30T14:57:48Z
dc.date.created2016-07-03T19:30:27Z
dc.date.issued2016
dc.identifier.citationAshworth, M. and Han, H. and Knell, G. and Powles, S. 2016. Identification of Triazine-Resistant Vulpia bromoides. Weed Technology. 30 (2): pp. 456-463.
dc.identifier.urihttp://hdl.handle.net/20.500.11937/42159
dc.identifier.doi10.1614/WT-D-15-00127.1
dc.description.abstract

In Australia, triazine herbicides have routinely controlled the Vulpia species (Vulpia bromoides, Vulpia myuros, and Vulpia fasciculata; collectively referred to as silvergrass). However, a simazine-resistant silvergrass biotype, collected from Pingelly in the Western Australian grain belt in 2014, has been confirmed. Compared to the pooled mortality of three simazine-susceptible silvergrass populations (S1, S2, and S3), the simazine-resistant Pingelly population was > 594-fold resistant at the LD50 level. Dose-response screening of the simazine-selected progeny (> 800 g ai simazine ha−1) demonstrated that the simazine resistance mechanism was heritable. Sequencing of the chloroplast psbA gene revealed the resistant population is homozygous for a serine 264 to glycine mutation, which confers a high-level triazine resistance. As expected this Ser-264-Gly mutation conferred resistance to atrazine and metribuzin, but not the phenyl-urea diuron. This is the first published report confirming field-evolved triazine resistance in a Vulpia population.

dc.publisherWeeds Science Society of America
dc.titleIdentification of Triazine-Resistant Vulpia bromoides
dc.typeJournal Article
dcterms.source.volume30
dcterms.source.number2
dcterms.source.startPage456
dcterms.source.endPage463
dcterms.source.issn0890-037X
dcterms.source.titleWeed Technology
curtin.departmentCentre for Crop Disease Management
curtin.accessStatusFulltext not available


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