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dc.contributor.authorWu, M.
dc.contributor.authorFalasca, Marco
dc.contributor.authorBlough, E.
dc.date.accessioned2017-01-30T15:10:17Z
dc.date.available2017-01-30T15:10:17Z
dc.date.created2015-10-29T04:09:56Z
dc.date.issued2011
dc.identifier.citationWu, M. and Falasca, M. and Blough, E. 2011. Akt/protein kinase B in skeletal muscle physiology and pathology. Journal of Cellular Physiology. 226 (1): pp. 29-36.
dc.identifier.urihttp://hdl.handle.net/20.500.11937/43812
dc.identifier.doi10.1002/jcp.22353
dc.description.abstract

The Akt/protein kinase B is critical regulator of cellular homeostasis with diminished Akt activity being associated with dysregulation of cellular metabolism and cell death while the regulation of Akt function has been well characterized in vitro, much less is known regarding the function of Akt in vivo. Here we examine how skeletal muscle Akt expression and enzymatic activity are controlled, the role of Akt in the regulation of skeletal muscle contraction, stress response glucose utilization, and protein metabolism, and the potential participation of this important molecule in skeletal muscle atrophy, aging, and cancer.

dc.titleAkt/protein kinase B in skeletal muscle physiology and pathology
dc.typeJournal Article
dcterms.source.volume226
dcterms.source.number1
dcterms.source.startPage29
dcterms.source.endPage36
dcterms.source.issn0021-9541
dcterms.source.titleJournal of Cellular Physiology
curtin.departmentSchool of Biomedical Sciences
curtin.accessStatusFulltext not available


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